Roberta Pelanda
Associate Professor
Research AreasResearch Areas
  • Autoimmunity/Rheumatology
  • Basic Immunology
  • Cellular and Molecular Biology

Special Interests

Research Interests

Immunology B cell biology Autoimmunity Signal transduction


University of Milan, Italy, PhD, Cellular and Molecular Biology
Max-Planck Institute for Immunobiology (Freiburg, Germany),
Institute for Genetics (Cologne, Germany),

Awards & Recognition

Harmon Foundation Award for Arthritis Research Arthritis Foundation, Rocky Mountain Chapter Grant

Professional Memberships



Velez M.G., Kane M., Liu S., Gauld S.B., Cambier J.C., Torres R.M., and Pelanda R., 2007. Ig allotypic inclusion does not prevent B cell development or response, J Immunol, 179:1049-1057.

Liu S., Velez M.G., Humann J., Rowland S., Conrad F.J., Halverson R., Torres R.M., and Pelanda R., 2005. Receptor editing can lead to allelic inclusion and development of B cells that retain antibodies reacting with high avidity autoantigens. J Immunol, 175:5067-5076.

Halverson, R,. R.M. Torres, and R. Pelanda, 2004. Receptor editing is the primary mechanism of B cell tolerance toward membrane antigens. Nature Immunology, 5(6):645-50.

Pelanda R., U. Braun, E. Hobeika, M.C. Nussenzweig, and M. Reth. 2002. B Cell Progenitors Are Arresred in Maturation but Have Intact VDJ Recombination in the Absence of Ig-a and Ig-b. J Immunol, 169:865-872.

Pelanda, R., S. Schwers, E. Sonoda, R.M. Torres, D. Nemazee, and K. Rajewsky. 1997. Receptor editing in a transgenic mouse model: site, efficiency, and role in B cell tolerance and antibody diversification. Immunity 7, no. 6:765-75.

Academic Affiliations

Associate Professor of Immunology, University of Colorado Denver

Contact Information

  • Office: 303.398.1451
  • Fax: 303.270.2325
  • Email:



  • National Jewish Health Main Campus
    1400 Jackson St.
    Denver, CO 80206

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