Peter M. Henson, MD, PhD, BVMS Ask a Question Refer Patient Peter M. Henson, MD, is a researcher at National Jewish Health. Dr. Henson is in the Division of Cell Biology and Department of Pediatrics. Professor Department of Immunology and Genomic Medicine Department of Pediatrics Division of Cell Biology Castle Connolly & 5280 Top Doctors 2023 Previously a 5280 Top Doctor America’s Top Doctors 2015 — Castle Connolly, Medical, Ltd. Recognized in America’s Top Doctors — Castle Connolly, Inc. Best Doctors in America® 2019-2020 — Best Doctors, Inc. Recognized in Best Doctors in America® — Best Doctors, Inc. America’s Top Doctors 2020 — Castle Connolly Medical, Ltd. Email Profile Print Profile Overview Contact Info & Locations Programs & Services Cell Biology Research Areas Basic Immunology Cellular and Molecular Biology Inflammation Lung Cell Biology Mycobacterial and Respiratory Infections Pathology Pulmonary Medicine Regenerative Medicine Allergy Special Interests Innate immunity, experimental pathology, Inflammation and inflammmatory respiratory diseases See More View Curriculum Vitae View Publications Education Education 1964 - 1967 University of Cambridge, PhD 1963 - 1964 University of Edinburgh, BSc with Honors, Microbiology 1958 - 1963 University of Edinburgh, BVM&S Fellowship 1967 - 1969 Scripps Research Institute, Experimental Pathology Awards & Recognition 2011: Distinguished Professorship, University of Colorado 2005: Burns Amberson Lecture, ATS Centenary Meeting 1991: Margaret A. Regan Professor of Pulmonary Inflammation 1983: Reticuloendothelial Society, Marie T. Bonazinga Award 1980:American Association of Pathologists, Parke Davis Award Publications Fadok VA, Voelker DR, Campbell PA, Cohen JJ, Bratton DL and Henson PM. Exposure of phosphatidylserine on the surface of apoptotic lymphocytes triggers specific recognition and removal by macrophages. J Immunol. 148(7):2207-2216, 1992. Fadok VA, Bratton DL, Konowal A, Freed PW, Westcott JY, Henson PM. Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGFβ, PGE2, and PAF. J. Clin. Invest. 101:890-898, 1998. Huynh, M-LN, Fadok VA, Henson PM. Phosphatidylserine-dependent ingestion of apoptotic cells promotes TGFβ1 secretion and resolution of inflammation. J Clin Invest 109:41-50, 2002. Gardai S.J, McPhillips KA, Frasch SC, Janssen W.J, Starefeldt A, Murphy-Ullrich JE, Bratton DL, Oldenborg PA, Michalak M, Henson PM. Cell-surface calreticulin initiates clearance of viable or apoptotic cells through trans-activation of LRP on the phagocyte. Cell 123:321-334, 2005. Gardai SJ, Xiao Y-Q, Dickinson M, Nick J, Voelker D, Greene K, Henson P. By binding SIRPα or calreticulin/CD91, lung collectins act as dual function surveillance molecules to suppress or enhance inflammation. Cell. 115:13-23, 2003. Desch AN, Gibbings SL, Clambey ET, Janssen WJ, Slansky JE, Kedl RM, Henson PM, and Jakubzick C. Dendritic cell subsets require cis-activation for cytotoxic CD8 T-cell induction. Nature communications, 5:4674 2014. Academic Affiliations Professor, Departments of Immunology and Microbiology, Medicine, Pharmacology, University of Colorado Denver Teaching & Professional Positions Associate Director, Pulmonary and Critical Care Fellowship Program Member, Immunology Graduate Program Steering Committee Ask a Question through MyChart Sign in to your MyChart account to communicate with your care team, manage appointments, and more. Create an Account Contact Information Office: 877.225.5654Fax: 303.398.1381 Patient Ratings The Patient Rating score is an average of all responses to care provider related questions on our independent rating system, the Press Ganey Patient Satisfaction Survey. This survey is about the patient care experience and does not address crucial characteristics like medical decision-making, prescribing the best therapy, and patient outcomes. Responses are measured on a scale of 1 to 5, with 5 being the best score. Learn more about our patient satisfaction survey. Comments Comments are collected in our Press Ganey Patient Satisfaction Surveys. Patients are de-identified to protect confidentiality and patient privacy. Learn more about our patient satisfaction survey.